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Assays of GBM cells stably transfected to over-express galectin-1, perfectly fit in with the previous studies mentioned above and highlight the importance of galectin-1 in the biologically aggressive behavior of experimental GBMs. While there was no enhancement of proliferation or change inattachment to fibronectin, galectin-1 upregulation induced more rapid two-dimensional migration and enhanced
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T least one of these symptoms was said to be bothersome,Hemminki et al. BMC Women's Health 2012, 12:44 http://www.biomedcentral.com/1472-6874/12/Page 3 ofthe woman was categorized as having bothersome symptoms of that group.Table 2 Proportions ( ) of women with vasomotor symptoms and bothersome symptoms1 in past two weeks, at each survey (cross-sectional analysis)Recruitment Women
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Ental counterparts. We did not observe, however, distant invasion in U87MG tumors over-expressing galectin-1. The U87MG model is in fact weakly invasive in the brains of immunocompromized mice [33,34], while it is associated with pronounced neoangiogenesis processes [37]. Further work (e.g. viral transduction) with our patient-derivedToussaint et al. Molecular Cancer 2012, 11:32 http://www.molecul
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Ional abuse behavior, against the women attending hospitals in Erbil. Physical violence is also a significant problem particularly in terms of its consequences.Background Violence against women is a worldwide problem and a serious human rights abuse that occurs among all social, cultural, economic, and religious groups. A wide range of negative health outcomes and even death had been recognized as
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Ional abuse behavior, against the women attending hospitals in Erbil. Physical violence is also a significant problem particularly in terms of its consequences.Background Violence against women is a worldwide problem and a serious human rights abuse that occurs among all social, cultural, economic, and religious groups. A wide range of negative health outcomes and even death had been recognized as
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Ble cross-hybridizing host genes. The use of our animal model to identify mediators of glioma invasion has the potential pitfall of identifying artifacts of xenografting. That is, human glioma cells confronted with nude mouse brain rather than human brain may express genes specific to this setting. Two arguments can be made against this theory. First, there is no teleological reason for human cell
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Duced CD55 promotes T-cell proliferation and Th1 cytokine expression. In addition to C3 production, APCs cleave C3 leading to autocrine and paracrine C3R signaling. C3R signaling promotes MHC class II expression, IL-12 production and B7 co-stimulatory molecules. Dendritic cells that fail to express C3aR suffer reduced T-cell activation. Anaphylatoxins are well known initiators of inflammation but
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Duced CD55 promotes T-cell proliferation and Th1 cytokine expression. In addition to C3 production, APCs cleave C3 leading to autocrine and paracrine C3R signaling. C3R signaling promotes MHC class II expression, IL-12 production and B7 co-stimulatory molecules. Dendritic cells that fail to express C3aR suffer reduced T-cell activation. Anaphylatoxins are well known initiators of inflammation but