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D the study, interpreted data, and prepared the manuscript. All authors read and approved the final manuscript. Acknowledgements This work was supported in part by grant number 1RO1 ES09882 from the National Institute of Environmental Health Sciences. Author Details 1Penn State Center for Host defense, Inflammation, and Lung Disease (CHILD) Research, Hershey, PA 17033, USA, 2Department of Pediatri
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Ex and exacerbating tooth decay. They have been associated with mutations in collagen in cases of osteogenesis imperfecta (OI) [136]. Defects in the human DSPP gene are the major cause of dentin disorders identified to date [137,138,139]. Mutations in DSPP are associated with five different types of inherited dentin defects ?dentinogenesis imperfecta (DGI) types I, II, III and dentinal dysplasias
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Ex and exacerbating tooth decay. They have been associated with mutations in collagen in cases of osteogenesis imperfecta (OI) [136]. Defects in the human DSPP gene are the major cause of dentin disorders identified to date [137,138,139]. Mutations in DSPP are associated with five different types of inherited dentin defects ?dentinogenesis imperfecta (DGI) types I, II, III and dentinal dysplasias
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Th Gag(p17)-E2 particles induced antigen-specific cytotoxic [65] CD8+ T cellsTh Gag(p17)-E2 particles induced antigen-specific cytotoxic [65] CD8+ T cells able to produce IFN- . Moreover, a moderate neutralizing antibody response was found in rabbits immunized with an E2 scaffold displaying a peptide [67] mimotope of the HIV-1 gp120 bridging sheet . Furthermore, E2 multimeric scaffolds displ
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Ompared to WT mice [12,15], suggesting that, at least with respect to K. pneumoniae, direct effects of SPA on host defense function (i.e. enhancement of phagocytosis) are more important than the pleiotropic indirect effects on the BAL proteome.8. 9.10.Additional materialAdditional file 1 MIAPE: Gel Electrophoresis. File containing Minimum information about a proteomics experiment - Gel Electrophor
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